最後更新：2026年10月1日
血糖<60 mg/dL引起中樞神經症狀。常見於胰島素過量、敗血症、肝衰竭。需立即補充葡萄糖。。Metabolic pathophysiology involves disruption of normal biochemical pathways affecting energy metabolism, electrolyte balance, or acid-base homeostasis。
Feline hepatic lipidosis develops rapidly (2-7 days of anorexia) due to high obligate protein requirements and fat mobilization. Obese cats are at highest risk. Aggressive caloric supplementation via feeding tube is lifesaving. Nutritional secondary hyperparathyroidism may occur in cats fed all-meat diets.
Exertional rhabdomyolysis may occur in working or exercised dogs, presenting with myoglobinuria and elevated CK. Breed-specific metabolic disorders include cystinuria (Newfoundland, Labrador) and hyperuricosuria (Dalmatian). Nutritional secondary hyperparathyroidism may occur with calcium-deficient diets.
這頁有幾個欄位是同系統共用的模板，還沒針對本疾病單獨改寫過。請以臨床判斷為準，記得對照下方文獻。
建議：依代謝異常調整：含 Dextrose（低血糖），含 KCl（低血鉀）
監測頻率：q4-8h（急性期）
[1]Efficacy and Safety of Non-Insulin Antidiabetic Drugs in Cats: A Systematic Review.
[2]Current understanding on pathogenesis and effective treatment of glycogen storage disease type Ib with empagliflozin: new insights coming from diabetes for its potential implications in other metabolic disorders.
[3]Treatment and prevention of severe hypoglycaemia in people with diabetes: Current and new formulations of glucagon.
[4]Hypoglycemia in dogs: Causes, management, and diagnosis.
病情可能在 24-48 小時內惡化，需盡快評估與處置 (may deteriorate within 24-48h, prompt evaluation needed)
飼主衛教
用藥指引
⚠ 警訊症狀（立即回診）
環境調整
長期計畫：慢性代謝病每 3 月追蹤
適度活動
適度活動
期間：穩定後
及時補充葡萄糖預後良好。延遲可能腦損傷。
低血糖急症 診斷流程
詳細問診包括發病經過、用藥史、飲食與環境
CBC、生化全套、尿液分析
來源：自動產生